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What causes arrhythmia? Are there too many things to put down? I'm sure most people come to this page, like me, because they are experiencing arrhythmia and are wondering what to do. Shouldn't there be sections here directed at the concerned layman explaining what symptoms warrant a visit to the doctor, what causes it, what triggers it, and so on. Kimholder 03:51, 28 March 2007 (UTC)
I was wondering what the relationship between antipsychotic medications with Sudden Cardiac Death warnings and Death from Cardiac dysrythmia is. Do these medications include dysrythmia in the warning? any idea whould be helpful. —Preceding
unsigned comment added by
174.29.164.153 (
talk)
20:11, 19 September 2010 (UTC)
Do primates experience arrhythmia in the same way that humans do? Has there been any research into arrhythmia experienced by chimpanzees, baboons, or gorillas? — Preceding unsigned comment added by 75.164.247.233 ( talk) 20:34, 19 October 2015 (UTC)
The etiologies ('causes') of arrhythmia should be distinguished from the pathophysiologies (mechanisms). That being said, neither of those aspects have been touched upon in this section (and they receive little treatment across the rest of the article, too). Ndea7941 ( talk) 13:08, 6 October 2021 (UTC)
Is the ICD9 code correct? 427.9 is, I think, too specific. Even the icd9.chrisendres.com site linked to calls all of 427.x 'Cardiac dysrhythmias'. -- user:Somej
Removed:
SADS, or sudden arrythmia death syndrome, is a rare condition blamed for some otherwise unexplained deaths. It is related to long QT syndrome, QT being the time taken by the heart to recover after each beat. Patients with long QT can be given beta-blocker drugs to slow the heartbeat, and are advised to refrain from competitive sports.
-> Plan to add a new page on long QT syndrome.
I cut the following cryptic bullet-point outline from the Diagnosis section. Some of them are now addressed (with explanation) in "Origin of impulse", but, as with most Powerpoint-style presentations detached from their lecture, I couldn't reconstruct the train of thought behind the rest of the bullets. I hope someone can add some of this content back in a way that will make sense to readers who aren't cardiologists. ← Hob 20:29, 2004 Sep 2 (UTC)
- Mode of initiation
- Automaticity
- Enhanced or abnormal
- Spontaneous onset of tachycardia
- No premature beats leading to the arrhythmia
- Gradual increase in the rate of the arrhythmia over the first 5-10 beats ("warm up")
- EKG appearance of the first tachycardia's beat is identical to the rest
- Reentry
- Initiation is with a premature beat followed by a slight pause
- This is followed by the arrhythmia (corresponding to premature beat, unidirectional block, slow conduction)
- "Warm up" is unusual
- EKG appearance of the first tachycardia beat need not be identical to the rest
- Mode of termination in response to overdrive pacing (pacing the heart at a rate faster than the tachycardia rate). Sometimes the application of electrical pacing from outside the heart itself is useful diagnostically. This is most commonly done by placing an electrode into the cardiac chambers and delivering electrical current across the endocardium
- Automaticity
- Often shows "overdrive suppression"
- The arrhythmia seems to be terminated by pacing only to return after several seconds with a gradual resumption of the pre-pacing rate
This is related to increased activity of the Na+ - K+ pump with Na+ loading. This causes the cell to have a more negative resting membrane potential and takes longer to reach threshold.- Reentry
- Often terminates in response to overdrive pacing
- Without subsequent arrhythmia resumption
Tachycardia stops because paced impulses have entered circuit in both limbs causing bi-directional block
(Also, I really can't figure out what this paragraph was trying to say:)
Cardiac dysrhythmia is technically more correct, as arrhythmia would imply that there is "no rhythm,"
In Greek "Rhythmia" means evidently "Rythm", (the Greek one being the original). So "Arrhythmia" indeed (not implied at all) means "no rhythm" as it is stated in the article.
But why this is not correct???. "No rhythm" doesn't mean "No pulse" or “No bit”. "Arrhythmia" means simply "pulse without rhythm".
Yet, I can still support the article’s suggestion, saying (as a Greek) that the word "Dysrtythmia" (Bad-rhythm) is also correct and identical in meaning (literature-wise - not aware if also medically). I guess that “Bad-rhythm” and “No-rhythm” is logically the same thing. I also guess that we could say “Mal-rhythm”..
-- 62.38.24.151 Dimitri GIANNAKOPOULOS / dimitri999@lycos.com / 00:20, 21 November 2005 (UTC)
I found this in the main article, so I transferred it to discussion:
" "In defibrillation, the recipient has lost consciousness so there is no need for sedation."
As someone who has recently had 17 defibrillations from an implanted unit, I can state, unequivocally, that it hurts like hell and the shock comes before there is any loss of consciousness. " Snodawg 22:32, 3 February 2006 (UTC) (P.S. I did not type this originally, merely transferred it)
I have sinus arrythmia and tried searching for it on here to see what we have and nothing on this page talks about it though it is one of the most common forms of arrhythmia for athletes and adolesence. I think it should be added.
Anomalycp 18:01, 26 April 2006 (UTC) I stand corrected from the opening paragraph, perhaps we need to change it to a subheading in the "common arrhythmias" section. --
User:Strolch1983 German wikipedia /
15:02, 27 March 2006 (UTC)
Hallo, if you are ever interested in writting an article for the reentry mechanism in english, you can link it, if you want, with the German wikipedia article "Kreisende Erregung". best greetings. --Strolch1983
I produced a PC based model that dynamically illustrated the mechanisms of reentry and fibrillation and other cardiac behaviour. I did this for my masters thesis in Biomed Engineering.
It allowed the student to set up the preconditions for the arrythmia and then watch it develop. I called it XCITE.
This was fifteen years ago. I don't know if it was ever picked up by educators. It was good.
I have been searching the web for information, yet finding anything in "laymans" terms is almost impossible! The Dr. told me that my "middle" chamber of my heart is not beating correctly with the top and bottom. His recomendation is an imediate pace maker! I can feel it skipping 1-3 beats some times and it feels like some one is inside my chest punching outward. then it will race and at other times will slow down, i get extremely tired, and at times get suddenly dizzy and will lose my eyesight aprox. 3-6 seconds. then the dizzyness is gone and my eyesight comes back. during and EEG and EKG and echo, the techs. told me they had never seen some one with so many skipped beats.
MY QUESTION; what are some possible reasons for this to happen? and why is there no photo of the heart showing each chamber and the flow of function.
Just reverted a change from last week that chopped about the first third of the article off... I'd be the first to admit there is a lot of work waiting to be done here, but axing the Definition, Manifestations and Mechanisms section probably didn't help its cause.
This article could use lotsof, some, any REFERENCES. The mechanism section needs copyediting... a lot of other arrhtymia articles (such as the VT and SVT) link to this bit. The treatment section needs expanding and linking. The chapter headings could be brought into line with the Manual of Style for Medical Articles And the whole thing needs to be looked at carefully by a non-doctor to de-jargon it.
I don't think it needs many more external links - Google gets you more university and medical school sites than you can poke a stick at! I'll keep chipping away at it from time to time... Happy editing. TamePhysician ( talk) 05:32, 29 March 2008 (UTC)
Would it be appropriate to add a section about SADS in the media? Various police agencies have blamed the death of prisoners/arestees on "Sudden Adult Death Syndrome"
Loimprevisto ( talk) 14:12, 3 September 2008 (UTC)
I am starting on a Zio Patch heart monitor. It's similar to a Holter Monitor, but you use it for up to two weeks of dat a collection. The Zio Patch was a 2010 winner of the Medical Design Excellence Awards (MDEA) [1] and here is more about the Zio Patch [2] This is my first time using a wiki talk page, apologies if I didn't format correctly.
Edsregistry ( talk) 22:09, 28 January 2014 (UTC)
References
There is only one arrhythmia it is asystole. The rest are dysrhythmias. The name of this page should be changed to reflect that. Doc James ( talk · contribs · email) 16:13, 11 October 2009 (UTC)
I noticed that 2 different mechanisms for causing dysrhythmias are missing - bypass tracts, where the impulse loops around the heart in abnormal ways, due to birth defects etc (see Wolff-Parkinson White Syndrome). The second is conduction blocks, see heart blocks for this. These should be added by someone who knows fully about it, I'm not confident enough to add the section (and have exams now :P) FruitywS ( talk) 13:40, 23 April 2010 (UTC)
I pasted the following analogy of reentry to here, because some issues need fixing: Actually, even in the normal case, people would see other people standing up when they sit down after 3 seconds. A functional analogy could be to sit down and close the eyes for 3 seconds to avoid seeing other people standing up. But at this level it seems to be as complicated as what goes on in nerves, so an analogy seems of little help. 193.235.173.250 ( talk) 12:51, 7 May 2010 (UTC)
By analogy, imagine a room full of people all given these instructions: "If you see anyone starting to stand up, then stand up for three seconds and sit back down." If people are quick enough to respond, the first person to stand will trigger a single wave which will then die out; but if there are stragglers on one side of the room, people who have already sat down will see them and start a second wave, and so on.
Does the above have a WP:MEDRS to cite? LeadSongDog come howl 13:51, 7 May 2010 (UTC)
PMC 1502051 that editors here might find useful. LeadSongDog come howl! 17:06, 5 November 2010 (UTC)
As somebody who's just had to have an A-fib turned using electricity for the first time, with the problem most likely having been precipitated by my abuse of alcohol... From pure experience, I'm pretty much perfectly sure overuse of alcohol could be one of the things which causes sinus arrhythmia in adults, perhaps in conjunction with non-lifethreating, recurrent atrial fibrillation. Especially upon "sleeping it off". Earlier I've talked about my own experience by referring to "morning after hyperventilation", but now I think that's not it.
I haven't seen this one mentioned in either this article or the one about sinus arrhythmia, but after my most recent episode, it sounds like something that could/should be easily found in the literature as well. I can't write anything about the phenomenon, though, because I'm not in this field, so that I don't have access to the relevant research literature. Thus, I'd like to suggest somebody else around here could perhaps take a look at the addiction and cardiac research, to verify whether my intuition/anecdote pans out. After all, given the huge rate of alcohol abuse all over the world, and even my not having been able to find words for something that prolly forsaged a more serious arrhythmia, I'm pretty sure that this stuff should be on Wikipedia. I mean, if it proves to be a well-researched and easily citable result. Decoy ( talk) 21:22, 24 August 2011 (UTC)
Normal hearts are not perfectly rhythmic. Please add information about the details of the range of "normal" -- how many PACs, PVCs etc normal hearts have per 24 hrs, etc.- 96.237.13.111 ( talk) 14:12, 21 November 2011 (UTC)
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There is a sentence that contradicts itself: "SADS is used to describe sudden death due to cardiac arrest brought on by an arrhythmia in the absence of any structural heart disease on autopsy. The most common cause of sudden death in the US is coronary artery disease.[citation needed]" If there is an absence of any structural heart disease in autopsy, then why does it go on to say that the cause is coronary artery disease, which WOULD show as a structural heart problem on autopsy? Medical experts please enlighten and perhaps correct that sentence as it is very misleading. 58.167.196.2 ( talk) 03:29, 29 November 2012 (UTC)
Can this treatment be mentioned in the article ?: Gene Therapy for fixing heart rythm disorder
I saw dear Doc James has redirected the article to current name (Cardiac Dysrhythmia) with this reasoning that arrhythmia means there is no rhythm at [1] but I think this is a personal reasoning and we should use terms that used in scientific literature. A simple googling can show that "arrhythmia" is using in medical literature for example by American heart association at here or you can see many book titles here or more important that MeSH has chosen "Arrhythmia" at here. Dysrhythmia is an old term that had been used in 1970s and 1980s. However dysrhythmia is used in cerebral context as "cerebral dysrhythmia" or "Thalamocortical dysrhythmia". I think we should change the title to "Arrhythmia" event without "cardiac" word. Baqeri ( talk) 15:52, 9 January 2015 (UTC)
Yes can move back to cardiac arrhythmia. The term arrhythmia is also applied to sleep such as Circadian arrhythmia Doc James ( talk · contribs · email) 20:32, 11 January 2015 (UTC)
@ Redrose64: Just wondering why the talk page continues to come up as Talk:Cardiac_dysrhythmia (?) even though the page name has returned to Arrhythmia. Cheers, 86.181.67.166 ( talk) 12:17, 14 January 2015 (UTC)
Thanks Doc James. It is true that the term "circadian arrhythmia" is used but it is not frequent and it is not registered in MeSH. However the term "arrhythmia" is used frequent for the meaning of "cardiac arrhythmia" for example in MedlinePlus. When We use "arrhythmia" We refer to "cardiac arrhythmia" for example see this Uptodate article or this. Baqeri ( talk) 10:00, 17 January 2015 (UTC)
Have moved this content here "Histamine modifies the heart rhythm and is responsible for many cardiac arrhythmias. The major arrhythmogenic actions of histamine are an H1-receptor-mediated slowing of atrioventricular conduction and H2-receptor-mediated changes in sinus rate and ventricular automaticity. Central nervous system H1-histamine receptors may mediate cardiac arrhythmias that occur after intracerebral hemorrhage or similar intracranial insults. Histamine, via the H2-receptor, attenuates the tachycardia produced by stimulation of cardiac sympathetic nerves or exogenous norepinephrine. H2-receptor blockade in this setting might therefore be expected to exacerbate ventricular arrhythmias secondary to loss of the histamine-mediated attenuation of sympathetic activity. The weight of clinical and experimental data justifies a trial of H1 and H2 receptor blockers when conventional antiarrhythmic therapy fails in the treatment of life-threatening cardiac arrhythmias in a clinical setting suggestive of histamine release. [1]"
We should use something newer than 1986. Doc James ( talk · contribs · email) 21:16, 10 January 2015 (UTC)
I'm moving [5] the following paragraph from the main page due to concerns about weighting (as well as the need for recent sourcing), and the way some of it is framed under "==Differential diagnosis=="[?]
Histamine modifies the heart rhythm and is responsible for many cardiac arrhythmias. The major arrhythmogenic actions of histamine are an H1-receptor-mediated slowing of atrioventricular conduction and H2-receptor-mediated changes in sinus rate and ventricular automaticity. [2] Also, some antihistamines appear to increase the risk of dysrhythmias. [3]
86.181.67.166 ( talk) 09:14, 13 January 2015 (UTC)
I would like to share information about CANet (ideally with an article of its own), but I have a COI as I am an employee of the organization. It is one of 13 currently-funded Networks of Centres of Excellence - which are research-based networks that require multi-disciplinary (e.g. academic, industry, government) cooperation and multi-centre cooperation to fulfill their mandates. [1] This article announced CANet's funding, and discusses some of the network's goals: http://london.ctvnews.ca/26-3m-for-canadian-arrhythmia-network-at-western-1.2148623. NKC717 ( talk) 19:14, 12 November 2015 (UTC)
For this. It does not fit here IMO. The table we currently have at antiarrhythmic drugs is better than this one. This one tries to put to much info into a table. This content is better dealt with as prose and within the articles about the medications in question. Doc James ( talk · contribs · email) 18:55, 11 March 2017 (UTC)
Extended content
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***Below is a table summarizing the drugs utilized in the treatment of cardiac arrhythmias. The table is currently undergoing development as a research project at the University of California, Los Angeles (UCLA). Expected date of completion is March 26, 2017 at 11:59 PM PST.
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This article has been at "heart arrhythmia" for about a year. Probably should have a move discussion before changing it again. Mayo uses the term [6]. Often just called "arrhythmia" but as there are other types good to have a qualifier. Doc James ( talk · contribs · email) 23:55, 14 May 2018 (UTC)
The result of the move request was: moved as requested per the discussion below. Dekimasu よ! 15:09, 9 December 2019 (UTC)
Heart arrhythmia → Arrhythmia – Arrhythmia can only relate to the heart and is the overwhelmingly name usually used. Google hits 11 million plus, ngrams clear highest usage of the three Iztwoz ( talk) 10:14, 2 December 2019 (UTC)
"While most types of arrhythmia are not serious..."
Would it be better to say most cases are not serious? Overwhelmingly presence of arrhythmia is not a serious issue. But saying most types are not serious, this to me implies some process of counting up the different types and asking which are "serious" and which not. There are however many types of serious arrhythmias that are very rare.
Edinburghpotsdam ( talk) 18:14, 14 July 2021 (UTC)
The description of trigeminy on this page is not quite correct.
The page:
PVCs that occur at intervals of 2 normal beats to 1 PVC are termed "PVCs in trigeminy"
In fact 2 normal to 1 PVC *or* 1 normal to 2 PVC are both considered trigeminy.
One source: https://www.healthline.com/health/trigeminy
Propose to add the second category to this sentence.
Edinburghpotsdam ( talk) 02:15, 17 July 2021 (UTC)
The description of the relationship between PVC triplets and VT is better described on the current Bigeminy page as well as having a good source:
I'm going to replace the same bullet point on the main arrhythmia page:
With the above text edited slightly. I don't know if this text really needs to be on the bigeminy-specific page so if it stands on the arrhythmia page I will take it off of there to avoid repetition.
Edinburghpotsdam ( talk) 19:22, 8 August 2021 (UTC)
The section as titled above ('Mechanism') has a single sentence - it is rather uninformative. I propose that it should be removed. Ndea7941 ( talk) 13:18, 6 October 2021 (UTC)